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Tyler and Family Medical Issues

Page history last edited by Henry T. Hill 8 months, 2 weeks ago

Ty - Gall Stones, Kidney Stones and Fatty Liver - Told 2/23/2023


Ty got CT or MRI  for kidney pains Wednesday, 2/22/2023


Ty called wit CT or MRI test results: many small kidney stones, gall stones and Fatty Liver. Thursday, 2/23/2023



Information About Fatty Liver Disease  from Gastrointestinal Society of Australia 

What causes fatty liver? Fatty liver is not caused simply by eating fatty foods. It is associated with health problems such as: Most commonly • Obesity (about 20% of people considered obese have fatty liver disease) • High blood cholesterol and triglycerides • Type 2 diabetes mellitus • Heavy alcohol use Less common causes • Underactive thyroid • Certain drugs • Polycystic ovary syndrome • Complications late in pregnancy


Nonalcoholic Fatty Liver - 2022

The most important complications of non-alcoholic fatty liver disease (NAFLD) in the descending order are:

  • Cardiovascular disease
  • Hepatocellular carcinoma
  • End-stage liver disease


  • Stage 0: No fibrosis
  • Stage 1: Zone 3 perisinusoidal fibrosis only
  • Stage 2: Zone 3 perisinusoidal and periportal fibrosis
  • Stage 3: Bridging fibrosis
  • Stage 4: Cirrhosis


Pathogenesis and Therapeutic Strategies Related to Non-Alcoholic Fatty Liver Disease - 2022 with 19 pages. Lists experimental treatments.



Pathogenesis and Therapeutic Strategies Related to Non ...

.Although patients with mild NAFL are considered to show no obvious clinical symptoms, patients with long-term NAFL may culminate in NASH and further liver fibrosis. Even though various drugs are able to improve NAFLD, there are no FDA-approved medications that directly treat NAFLD. 





Chernobyl - April 26, 1986


What kind of radiation were the people exposed to at Chernobyl?

Radiation released during the Chernobyl accident. Workers and the public were exposed to three main types of radionuclides: iodine-131, cesium-134 and cesium-137. When iodine-131 is released into the environment, it is quickly transferred to humans and taken up by the thyroid gland. Mar 9, 2022


Can you get radiation from another person Chernobyl? 

Radiation cannot be spread from person to person. Small quantities of radioactive materials occur naturally in the air, drinking water, food and our own bodies. People also can come into contact with radiation through medical procedures, such as X-rays and some cancer treatments.


  1. Lynn Miles  https://thechart.blogs.cnn.com/2011/03/23/health-lessons-from-chernobyl/comment-page-1/

    How sad... 
    - No, how criminal. You just might be duping people to their deaths, y'know.
    If we want truth in reporting, guess we'lll have to turn off our TVs and go to the Salt Lake Tribune....
    Written by Russian and Belarus experts, edited and published by the New York Academy of Sciences, the book is Chernobyl : Consequences of the Catastrophe for People and the Environment. Drawing from more than 5,000 published articles and studies, the authors’ conclusions about the disaster in the former Soviet Union are authoritative and startling. So far, 1 million people from around the world have already died from Chernobyl radiation, including over 110,000 of the original 830,000 cleanup workers. High doses of radioactive fallout reached much of Europe and the United Kingdom and 750 million people in the Northern Hemisphere received significant contamination. The accident released 200 times more radiation than previously thought, hundreds of times more than the bombs dropped on Hiroshima and Nagasaki.

    The actual number cited in the book is 985,000 deaths by 2004, so we extrapolate forward from that to arrive at OVER A MILLION DEATHS. And the suffering still goes on and on. This number is not put out by anti-nuke people who the CNN loves to deride as quacks, but by the very reputable NY Academy of Sciences - all but ignored by the mainstream press, not to mention apparently unknown to "experts" like Davis, who tell us the "high range" of the death estimates is "tens of thousands."

    CNN = Criminally Negligent Nukesters

    March 27, 2011 at 18:24 | Report abuse | Reply



  1. K Ulriksen

    I had a friend who worked on monitoring station for Chernobyl before it blew. His family and him lived in Kiev. While their older child, a daughter, doesn't have many problems, their son who was an infant at the time of the meltdown has bone's and teeth that are extremely brittle. At 8 years old, he had to be fitted for a full set of dentures. So they didn't die, but their lives were definitely negatively impacted by the incident. As some one who worked on the monitoring, he knew that the truth was being told about the extent of the damage. Consequently, he immigrated to the U.S.

    March 23, 2011 at 22:26 | Report abuse | Reply


Nuclear Accident Crisis and Liver Disease: A Summary on Evidences


International research teams explore genetic effects of Chernobyl radiation


NIH - National Cancer Institute Posted: April 22, 2021 


Reproductive Effects of Exposure to Low-Dose Ionizing Radiation: A Long-Term Follow-Up of Immigrant Women Exposed to the Chernobyl Accident

2020 Journal of Clinical Medicine Ben Gurion University Full text


Chronic Contamination with 137Cesium in Rat: Effect on Liver Cholesterol Metabolism 

effect on humans and liver, five pages, International Journal of Toxicology


Chernobyl Caesium-137

 More than twenty years after the Chernobyl accident, attention focuses mainly on one particular radioactive nucleus : caesium-137. Iodine-131, dreadful in the weeks following the disaster, has disappeared because of its 8 days radioactive half-life (or period). On the contrary, because of their periods of thirty years, the effects of cesium-137, and to a lesser degree those of one other radioactive element, strontium-90, are still being felt.

 The thirty years caesium half-lfe offers an advantage: it is 1400 times less active than iodine-131. But it carries a drawback: it disappears very slowly.

 A French respected intitution, the IRSN, estimates that that 80,000 cesium-137 terabecquerels were released into the environment, i.e. 30 to 40% of the amount present in the the damaged reactor core. The Becquerel (Bq) is a very small unit. One uses instead the TBq unit (one million of million of Bq) to represent the Chernobyl releases.

 Caesium had disappeared since a long time from the atmosphere after its deposition on the ground. Almost all deposits are found in shallow depth, because migration of this chemical element with minerals is low. It slowly sinks into the ground. More than twelve years after the accident, caesium was concentrated in the top 5 cm of plant litter, contaminating mainly young wood roots and fungi. Near Chernobyl, the interception of radioactive dust and aerosols by the foliage and then the leaf drop, led to a localized contamination of forest litter on an area of about 40 000 km2. In some areas near the plant, a few centimeters at the surface were removed or covered with uncontaminated soil. This cleanup has reduced the amount of cesium by a factor of 10 to 100.

 Caesium contamination, carefully followed by radiation protection agencies, has been the subject of 60 000 measurements since the accident. One observes leopard skin patches of contaminations that follow the pattern of iodine-131 spread in 1986.


Risk of Solid Cancers following Radiation Exposure: Estimates for the UK Population 270 pages, 2011


Page 114 and following

3.7 Liver 3.7.1 General epidemiology Liver cancer is the third most common cause of cancer death worldwide, but is relatively rare in the UK (Parkin et al, 2005). Liver cancer is highly fatal, but the mortality data are unreliable because metastatic cancers from other organs are often misclassified as liver cancer at death certification. Only a little over 50% of liver cancers reported on death certificates in the USA are attributable to primary liver cancer (Percy et al, 1990). Misclassified diagnoses can either underestimate or overestimate the association with radiation exposure, depending upon the organs from which cancer metastases occurred. The two major types of primary liver cancer are liver cell (or hepatocellular) carcinoma (HCC) and cholangiocarcinoma, which differ in aetiology and pathology. In the UK, there have been reported 30–50% increases in overall liver cancer mortality and incidence rates, with a more marked increase in cholangiocarcinoma, in the last two decades (West et al, 2006). Liver cell carcinomas are the predominant type of liver cancer in many populations and attributable largely (80–95%) to infection with hepatitis B or C virus (Parkin, 2006). Worldwide, hepatitis B virus infection is more prevalent than hepatitis C viral infection, except for Japan where liver cancers are mostly attributable to hepatitis C viral infection (Tanaka et al, 1994; Parkin et al, 2005). Excessive alcohol consumption is an acknowledged cause of HCC (IARC, 1988), but how alcohol consumption interacts with hepatitis viral infection in the pathogenesis of liver cell carcinoma is not well understood. Aflatoxin, a product of the fungus Aspergillus species, is an important cause of liver cancer in geographical areas where contamination of food with this toxin is common. Cholangiocarcinomas originating from the intrahepatic bile duct are likely to be similar to cancer of the extrahepatic bile duct and gallbladder cancer in their aetiology, and are more frequent in females than in males. High rates of cholangiocarcinoma have been linked to the endemic occurrence of liver fluke infection in certain areas of Thailand (Parkin et al, 2005; Thomas et al, 2006).











Environmental toxin-induced acute kidney injury

In particular, environmental toxins, to which we are unintentionally exposed via oral, inhalational or transdermal routes, are a common yet underappreciated cause of kidney injury. The high vulnerability of the kidney can be explained by its main physiological features, such as the highest blood flow per 100 g tissue, the largest endothelial surface by weight, highly active multiple metabolizing enzyme systems, the high concentration of filtered chemicals in tubular fluid adjacent to tubular cells, protein unbinding of chemical compounds in the tubules and further intrarenal biotransformation of chemicals. 

Aristolochic, Melamine, Metal-induced Nephropathy, Cadmium, Lead and Mercury.


In contrast to the majority of disease aetiologies, identifying the precise cause and mechanism of environmentally induced renal injury remains a daunting task involving many scientific disciplines, as evidenced above. Investigations in this field are particularly challenged by the apparent infinite types of toxins, their mutual interaction and handling/metabolization by the body.


Kidney and heavy metals - The role of environmental exposure (Review)

Lithium, Cisplatin,  Cadmium, Arsenic, Mercury and Lead.


Mysterious kidney disease traced to plant toxins

One plant examined in this article. 

The plant, Aristolochia clematitis—more commonly known as birthwort—can surely be called beautiful, but within it lurks a poison. Over the past few decades, researchers have identified a family of compounds in the plant, collectively called aristolochic acids (AAs), as the cause of a severe kidney disease endemic to this region, as well as the cause of multiple types of cancer.



Factitious Disorders (FDIA)


How do you prove FDIA?


According to DSM 5, the following criteria must be met in order to make the diagnosis of FDIA:

  1. The Perpetrator engages in the deceptive falsification of physical or psychological signs or symptoms, or of induction of injury or disease in another;
  2. The Perpetrator presents the victim to other as ill, impaired or injured;


What is an example of a somatic delusion?


Some examples of common somatic delusions include: Fears of infestation or infection. People with somatic delusions often believe they've been infected by parasites that have taken over their internal organs, or by tiny insects that have burrowed under their skin to lay their eggs. Distorted body image.




Factitious disorders 1 Early recognition and management of fabricated or induced illness in children 10 pages Lancet 2014


Personality disorder Very few studies have been done of personality in female perpetrators. In a systematic study of personality in 19 mothers with fabricated or induced illness (two-thirds with evidence of deception), high rates of personality disorder were identified in 17 (89%) mothers, with antisocial, histrionic, borderline, avoidant, and narcissistic categories being most frequently identifi ed.29 Some mothers had more than one personality disorder. In a German study, three of four mothers had a personality disorder, with paranoid personality diagnosed in two mothers.37 Borderline personality disorder in particular is associated with marked difficulties in mother–infant interaction.38


Munchausen's Syndrome and Other Factitious Disorders in Children


Factitious Disorder Imposed on Another (FDIA) Cleveland Clinic

Whether the false medical claims are about someone else or yourself, this condition can be dangerous. It can lead to unnecessary medical tests and even procedures while the healthcare team tries to figure out what’s wrong with the patient. When a child is involved, FDIA is considered a form of child abuse by the American Professional Society on the Abuse of Children.











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